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Literature record

Haemoglobinopathies, thalassaemias and enzymopathies in Saudi Arabia: the present status.

PMID 3120460 | DOI 10.1159/000205861 · Acta haematologica · 1987

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The presence of the sickle cell (Hb S) gene in Saudi Arabia was first reported by Lehmann et al. in 1963 [Nature 198, pp. 492-493]. Later, Hb S, alpha- and beta-thalassaemia, glucose-6-phosphate dehydrogenase deficiency and other enzymopathies were shown to occur at a variable prevalence in different regions of the country. Recent studies using restriction endonucleases have revealed alpha-globin gene arrangement and beta-globin gene polymorphism in the Saudi population. Interactions between abnormal genes are commonly encountered which often influence the clinical manifestations of sickle cell disease. In this paper, we present recent findings and discuss the status of haemoglobinopathies, thalassaemias and enzymopathies in Saudi Arabia.

Validated evidence

TypeEntitySource evidenceConfidenceExtractor
phenotypeglucose-6-phosphate dehydrogenase deficiency“Later, Hb S, alpha- and beta-thalassaemia, glucose-6-phosphate dehydrogenase deficiency and other enzymopathies were shown to occur at a variable prevalence in different regions of the country.”0.98phenotype_alias_lexicon_v2
phenotypesickle cell disease“Interactions between abnormal genes are commonly encountered which often influence the clinical manifestations of sickle cell disease.”0.98phenotype_alias_lexicon_v2
populationSaudi Arabia“Haemoglobinopathies, thalassaemias and enzymopathies in Saudi Arabia: the present status. The presence of the sickle cell (Hb S) gene in Saudi Arabia was first reported by Lehmann et al. in 1963 [Nature 198, pp. Recent studies using restriction endonucleases have revealed alpha-globin gene arrangement and beta-globin gene polymorphism in the Saudi population. In this paper, we present recent findings and discuss the status of haemoglobinopathies, thalassaemias and enzymopathies in Saudi Arabia.”0.95saudi_context_rules_v1